Frontiers | Mechanism of Endoplasmic Reticulum Stress in Cerebral Ischemia
Neuron‐specific translational control shift ensures proteostatic resilience during ER stress | The EMBO Journal
EGF-SubA enhances cytotoxic effects of the ER stress inducer... | Download Scientific Diagram
ER stress-induced cell death proceeds independently of the TRAIL-R2 signaling axis in pancreatic β cells | Cell Death Discovery
Endoplasmic reticulum stress stimulates the release of extracellular vesicles carrying danger-associated molecular pattern (DAMP) molecules | Oncotarget
Unfolded Protein Response (UPR) and Endoplasmic Reticulum (ER) Stress FAQs
Biomedicines | Free Full-Text | The Crosstalk of Endoplasmic Reticulum (ER) Stress Pathways with NF-κB: Complex Mechanisms Relevant for Cancer, Inflammation and Infection
ER stress-induced cell death mechanisms - ScienceDirect
What is Thapsigargin? Frequently Asked Questions
Effects of endoplasmic reticulum stress on apoptosis induction in radioresistant macrophages
Pancreatic INS-1 β-Cell Response to Thapsigargin and Rotenone: A Comparative Proteomics Analysis Uncovers Key Pathways of β-Cell Dysfunction | Chemical Research in Toxicology
Frontiers | STING, the Endoplasmic Reticulum, and Mitochondria: Is Three a Crowd or a Conversation?
Thapsigargin - Wikipedia
Cell death induced by the ER stressor thapsigargin involves death receptor 5, a non-autophagic function of MAP1LC3B, and distinct contributions from unfolded protein response components | Cell Communication and Signaling | Full Text
Endoplasmic reticulum stress, the unfolded protein response and autophagy in kidney diseases | Nature Reviews Nephrology
IJMS | Free Full-Text | An Involvement of Oxidative Stress in Endoplasmic Reticulum Stress and Its Associated Diseases
Endoplasmic Reticulum Stress Mediates Vascular Smooth Muscle Cell Calcification via Increased Release of Grp78 (Glucose-Regulated Protein, 78 kDa)-Loaded Extracellular Vesicles | Arteriosclerosis, Thrombosis, and Vascular Biology
MITOL prevents ER stress‐induced apoptosis by IRE1α ubiquitylation at ER–mitochondria contact sites | The EMBO Journal
Thapsigargin: key to new host-directed coronavirus antivirals?: Trends in Pharmacological Sciences
Endoplasmic Reticulum Stress Induces G2 Cell-Cycle Arrest via mRNA Translation of the p53 Isoform p53/47 - ScienceDirect
Phenotypic Discovery of Neuroprotective Agents by Regulation of Tau Proteostasis via Stress‐Responsive Activation of PERK Signaling - Shin - 2021 - Angewandte Chemie International Edition - Wiley Online Library
